Blood 1 5 O C T O B E R 2 0 0 8 I V O L U M E 1 1 2 , N U M B E R 8
نویسنده
چکیده
REFERENCES 1. Monroe DM, Hoffman M, Oliver JA, et al. Platelet activity of high-dose factor VIIa is independent of tissue factor. Br J Haematol. 1997;99:542-547. 2. Hedner U, Kisiel W. Use of human factor VIIa in the treatment of two hemophilia A patients with high-titer inhibitors. J Clin Invest. 1983;71:1836-1841. 3. Bom VJ, Bertina RM. The contributions of Ca2 , phospholipids and tissue-factor apoprotein to the activation of human blood-coagulation factor X by activated factor VII. Biochem J. 1990;265:327-336. 4. Rao LV, Rapaport SI. Factor VIIa-catalyzed activation of factor X independent of tissue factor: its possible significance for control of hemophilic bleeding by infused factor VIIa. Blood. 1990;75:1069-1073. 5. Rao LV, Rapaport SI. Cells and the activation of factor VII. Haemostasis. 1996;26 suppl 1:1-5. 6. HoffmanM,MonroeDMIII.A cell-basedmodelofhemostasis. ThrombHaemost.2001;85:958-965. 7. van’t Veer C, Golden NJ, Mann KG. Inhibition of thrombin generation by the zymogen factor VII: implications for the treatment of hemophilia A by factor VIIa. Blood. 2000;95:1330-1335. 8. Tranholm M, Kristensen K, Kristensen AT, et al. Improved hemostasis with superactive analogs of factor VIIa in a mouse model of hemophilia A. Blood. 2003;102: 3615-3620.
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